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Respuestas de inmunidad celular en la tuberculosis pulmonar.

Human pulmonary tuberculosis (TB) is a worldwide public health problem, which is caused by Mycobacterium tuberc-ulosis. It is a fact that one third of the world ' s population is infected with this mycobacteria, however , only a minority of people infected by M. tuberc-ulosis may develop a clinical...

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Udgivet: 2005
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Tuberculosis
mycobacterium tuberculosis
Alveolar macrophages
monocyte
macrophages
Online adgang:http://hdl.handle.net/10872/17525
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bitstream.checksumAlgorithm.fl_str_mv MD5
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bitstream.url.fl_str_mv https://saber.ucv.ve/jspui/bitstream/10872/17525/1/Investigacion.Clinica.2005.pdf
https://saber.ucv.ve/jspui/bitstream/10872/17525/2/license.txt
dc.contributor.author.none.fl_str_mv Araujo, Zaida
Vieyra- Reyes, Patricia
Rivas-Santiago, Bruno
dc.creator.fl_str_mv Araujo, Zaida
Vieyra- Reyes, Patricia
Rivas-Santiago, Bruno
dc.date.accessioned.none.fl_str_mv 2018-01-15T15:32:24Z
dc.date.available.none.fl_str_mv 2018-01-15T15:32:24Z
dc.date.issued.none.fl_str_mv 2005
dc.identifier.citation.en_US.fl_str_mv Araujo, Z. Et All. Respuestas de inmunidad celular en la tuberculosis pulmonar. Investigacion Clinica 46 (4) 391-412
dc.identifier.issn.none.fl_str_mv 0535-5133
dc.identifier.uri.none.fl_str_mv http://hdl.handle.net/10872/17525
dc.language.iso.en_US.fl_str_mv es
dc.publisher.en_US.fl_str_mv Investigacion Clinica
dc.subject.en_US.fl_str_mv Tuberculosis
mycobacterium tuberculosis
Alveolar macrophages
monocyte
macrophages
dc.title.en_US.fl_str_mv Respuestas de inmunidad celular en la tuberculosis pulmonar.
dc.type.en_US.fl_str_mv Article
description Human pulmonary tuberculosis (TB) is a worldwide public health problem, which is caused by Mycobacterium tuberc-ulosis. It is a fact that one third of the world ' s population is infected with this mycobacteria, however , only a minority of people infected by M. tuberc-ulosis may develop a clinical dis- ease. In general, about 90% have their bacilli under control in a latent state throughout their lives by means of their immune responses. About 5% will de- velop primary progressive TB and th<} remaining 5% will develop the disease in the later stages of their lives, which is known as reactivation or post-primary TB. In resistant individua1s, control of the infection mainly requires develop- ment of a Thl cell immunity response. This type of response involves participa- tion of a1veolar macrophages and T CD4 + , CD8 + and T yt5 Iymphocytes, and production of cytokines such as IL-2, IFN-y, IL-12, IL-18 and TNF-a, as well as chemokines such as MNTES, MCP-l, MIP-la and IL-8 which playan important role in the migration of different cell subpopulations to the infection site for the formation of granulome. In addition, the role of "natural killer" (NK) cells, a1ong with epitelial cells, is essentia1 as part of the innate immune response.
id SABERUCV_ee0e295dff701d0e2a055edee91c69dc
identifier_str_mv Araujo, Z. Et All. Respuestas de inmunidad celular en la tuberculosis pulmonar. Investigacion Clinica 46 (4) 391-412
0535-5133
language_invalid_str_mv es
network_acronym_str SABERUCV
network_name_str Repositorio Institucional de la Universidad Central de Venezuela
oai_identifier_str oai:saber.ucv.ve:10872/17525
publishDate 2005
publishDateSort 2005
repository.mail.fl_str_mv sfwrussians@gmail.com
repository.name.fl_str_mv Saber UCV
repository_id_str
spelling Araujo, ZaidaVieyra- Reyes, PatriciaRivas-Santiago, Bruno2018-01-15T15:32:24Z2018-01-15T15:32:24Z2005Araujo, Z. Et All. Respuestas de inmunidad celular en la tuberculosis pulmonar. Investigacion Clinica 46 (4) 391-4120535-5133http://hdl.handle.net/10872/17525Human pulmonary tuberculosis (TB) is a worldwide public health problem, which is caused by Mycobacterium tuberc-ulosis. It is a fact that one third of the world ' s population is infected with this mycobacteria, however , only a minority of people infected by M. tuberc-ulosis may develop a clinical dis- ease. In general, about 90% have their bacilli under control in a latent state throughout their lives by means of their immune responses. About 5% will de- velop primary progressive TB and th<} remaining 5% will develop the disease in the later stages of their lives, which is known as reactivation or post-primary TB. In resistant individua1s, control of the infection mainly requires develop- ment of a Thl cell immunity response. This type of response involves participa- tion of a1veolar macrophages and T CD4 + , CD8 + and T yt5 Iymphocytes, and production of cytokines such as IL-2, IFN-y, IL-12, IL-18 and TNF-a, as well as chemokines such as MNTES, MCP-l, MIP-la and IL-8 which playan important role in the migration of different cell subpopulations to the infection site for the formation of granulome. In addition, the role of "natural killer" (NK) cells, a1ong with epitelial cells, is essentia1 as part of the innate immune response.esInvestigacion ClinicaTuberculosismycobacterium tuberculosisAlveolar macrophagesmonocytemacrophagesRespuestas de inmunidad celular en la tuberculosis pulmonar.ArticleORIGINALInvestigacion.Clinica.2005.pdfInvestigacion.Clinica.2005.pdfapplication/pdf2396612https://saber.ucv.ve/jspui/bitstream/10872/17525/1/Investigacion.Clinica.2005.pdfb8d458abb80725c129a1e8b6f007ec1fMD51LICENSElicense.txtlicense.txttext/plain; charset=utf-81509https://saber.ucv.ve/jspui/bitstream/10872/17525/2/license.txt7c020282d630887ba819753d2e4a4c58MD5210872/17525oai:saber.ucv.ve:10872/175252018-01-15 15:32:24.96Saber UCVsfwrussians@gmail.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
spellingShingle Respuestas de inmunidad celular en la tuberculosis pulmonar.
Tuberculosis
mycobacterium tuberculosis
Alveolar macrophages
monocyte
macrophages
title Respuestas de inmunidad celular en la tuberculosis pulmonar.
title_full Respuestas de inmunidad celular en la tuberculosis pulmonar.
title_fullStr Respuestas de inmunidad celular en la tuberculosis pulmonar.
title_full_unstemmed Respuestas de inmunidad celular en la tuberculosis pulmonar.
title_short Respuestas de inmunidad celular en la tuberculosis pulmonar.
title_sort Respuestas de inmunidad celular en la tuberculosis pulmonar.
topic Tuberculosis
mycobacterium tuberculosis
Alveolar macrophages
monocyte
macrophages
url http://hdl.handle.net/10872/17525

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